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The year 1993 marked the discovery that mutations in the superoxide dismutase-1 (Sod-1) gene are responsible for causing Amyotrophic lateral sclerosis (ALS), and these mutations currently account for 18.9% of familial ALS cases.
Despite many years of scientific research, the underlying mechanisms of Amyotrophic lateral sclerosis (ALS) remain unclear. Most of the existing literature suggests that there are altered signaling pathways involved. These include oxidative stress, glutamate-induced excitotoxicity, neuroinflammation, and [mitochond…