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Helicobacter pylori infection
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Contents
Dysregulation of various cellular processes in the gastric epithelium and in different cells of the microenvironment is a hallmark of H. pylori infection
Myeloid-specific deletion of ornithine decarboxylase (ODC), the rate-limiting enzyme in polyamine synthesis, promoted M1 immune response and therefore significantly enhanced gastritis during Helicobacter pylori infection, but alleviated DSS-induced colitis
Helicobacter pylori infection upregulates NF-kB to induce inflammation in the stomach (Ya…
References (Sources)
- Helicobacter pylori Infection, the Gastric Microbiome and Gastric Cancer
- Ornithine Decarboxylase in Macrophages Exacerbates Colitis and Promotes Colitis-Associated Colon Carcinogenesis by Impairing M1 Immune Responses
- Ornithine decarboxylase regulates M1 macrophage activation and mucosal inflammation via histone modifications
- Relationship between Helicobacter pylori infection and Alzheimer disease
- The conundrum of Helicobacter pylori-associated apoptosis in gastric cancer