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Alzheimer's Disease (AD) & Pathogenicity
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Contents
Peripheral immune cells are crucial in the pathology of Alzheimer’s disease (AD), influencing key processes such as amyloid and tau protein aggregation, glial activation, neuronal integrity, and cognitive decline.
Amyloid-beta (Ab), tau, and Apolipoprotein E (APOE) are significant players in the pathogenesis o…
References (Sources)
- A unique microglia type associated with restricting development of Alzheimer’s disease
- Approaches for studying neuroimmune interactions in Alzheimer’s disease
- Astrocytes and oligodendrocytes undergo subtype-specific transcriptional changes in Alzheimer’s disease
- Genome-wide meta-analysis identifies new loci and functional pathways influencing Alzheimer’s disease risk
- Gram-negative bacterial molecules associate with Alzheimer disease pathology
- The Role of Reactive Oxygen Species in the Pathogenesis of Alzheimer’s Disease, Parkinson’s Disease, and Huntington’s Disease: A Mini Review
- Western diet as a trigger of Alzheimer’s disease: From metabolic syndrome and systemic inflammation to neuroinflammation and neurodegeneration