meRfi®-GM
The Digital Textbook on Gut Microbiota and Beyond
Mycobacterium tuberculosis
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Contents
Type I response induced by Mycobacterium tuberculosis strains is dependent on lysosomal transit
Interferons Type I (IFN Type I) expression correlates with the ability to induce pro-inflammatory responses
Oxysterol expression is associated with a higher pro-inflammatory response
Mycobacterium tuberculosis first encounters alveolar e…
References (Sources)
- ILC precursors differentiate into metabolically distinct ILC1-like cells during Mycobacterium tuberculosis infection
- Macrophage global metabolomics identifies cholestenone as host/pathogen cometabolite present in human Mycobacterium tuberculosis infection
- Microbiota and Tuberculosis: A Potential Role of Probiotics, and PostbioticsReview
- Modern Clinical Mycobacterium tuberculosis Strains Leverage Type I IFN Pathway for a Proiniammatory Response in the Host
- MR1-restricted mucosal associated invariant T (MAIT) cells in the immune response to Mycobacterium tuberculosis
- Mycobacterium tuberculosis produces D-serine under hypoxia to limit CD8(+) T cell-dependent immunity in mice.
- The role of cholesterol and its oxidation products in tuberculosis pathogenesis