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Mycobacterium tuberculosis
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Contents
Type I response induced by Mycobacterium tuberculosis strains is dependent on lysosomal transit
Interferons Type I (IFN Type I) expression correlates with the ability to induce pro-inflammatory responses
Oxysterol expression is associated with a higher pro-inflammatory response
see also:
[Exometabolites](brain://rcCsDRAr2Ea6HutRFFm…
References (Sources)
- ILC precursors differentiate into metabolically distinct ILC1-like cells during Mycobacterium tuberculosis infection
- Macrophage global metabolomics identifies cholestenone as host/pathogen cometabolite present in human Mycobacterium tuberculosis infection
- Modern Clinical Mycobacterium tuberculosis Strains Leverage Type I IFN Pathway for a Proiniammatory Response in the Host
- MR1-restricted mucosal associated invariant T (MAIT) cells in the immune response to Mycobacterium tuberculosis
- Mycobacterium tuberculosis produces D-serine under hypoxia to limit CD8(+) T cell-dependent immunity in mice.
- Mycobacterium tuberculosis–Induced Bronchoalveolar Lavage Gene Expression Signature in Latent Tuberculosis Infection Is Dominated by Pleiotropic Effects of CD4 + T Cell–Dependent IFN-g Production despite the Presence of Polyfunctional T Cells within the Airways
- The role of cholesterol and its oxidation products in tuberculosis pathogenesis