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NAFLD is associated with insulin resistance and insulin action disturbances, which promotes triglyceride accumulation (e.g., increased de novo lipogenesis).
In addition, hepatic insulin resistance could promote hepatocyte injury and inflammation which leads to NASH.
Insulin resistance seems to have a crucial role in the development of NAFLD by causing increased lipolysis at the adipocyte level, leading to an increased flux of non‐esterified fatty acids from adipose tissue to the liver.
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