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The etiology of allergies is intricately linked to type 2 inflammatory responses, which ultimately lead to the production of allergen-specific immunoglobulin E (IgE). This IgE is a key driver of many allergic conditions. At a high level, the initial exposure to an allergen disrupts epithelial integrity, triggering local inflammation through alarmins such as Interleukin-17E (IL-17E) / Interleukin-25 (IL-25), Interleukin-33 (IL-33), and [Thymic Stromal Lymphopoietin (TSLP)](brain://NyfDmiBFjUy4zxPQeyuxxg/ThymicStromalLymphop…