meRfi®-GM
Tau Pathology
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Contents
see also:
Alzheimer's Disease (AD) & Pathogenicity
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References (Sources)
- A genome-wide gene-expression analysis and database in transgenic mice during development of amyloid or tau pathology
- CCL2 overexpression in the brain promotes glial activation and accelerates tau pathology in a mouse model of tauopathy
- Mitophagy inhibits amyloid-β and tau pathology and reverses cognitive deficits in models of Alzheimer’s disease
- Transfer of a healthy microbiota reduces amyloid and tau pathology in an Alzheimer’s disease animal model