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Alzheimer’s disease (AD) is now much more than just an “amyloid‑tau proteopathy”: a chronically dysregulated immune response in the central nervous system (CNS) and the periphery is considered a core driver of disease development and progression. Neuroinflammation - especially through dysfunctional microglia and infiltrating peripheral immune cells - amplifies Amyloid-beta (Aß, Abeta) / Amyloid Plaques and [tau pathology](brain://yIOI2GVE0kOLV65yYLGR4Q/Ta…