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Experimental Colitis
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Contents
Fusobacterium nucleatum - Extracellular Vesicles (EV) significantly aggravated experimental colitis in mice associated with Fn-EV–mediated downregulation of miR-574-5p expression and autophagy activation
Blockade of autophagy using chloroquine alleviates barrier damage exacerbated by Fusobacterium nucleatum - Extracellular Vesicles (EV) in vitro and in vivo
Inhibition of the …
References (Sources)
- Acid sphingomyelinase inhibition suppresses lipopolysaccharide- mediated release of inflammatory cytokines from macrophages and protects against disease pathology in dextran sulphate sodium-induced colitis in mice
- Both Saccharomyces boulardii and Its Postbiotics Alleviate Dextran Sulfate Sodium-Induced Colitis in Mice, Association with Modulating Inflammation and Intestinal Microbiota
- Changes in serum inflammatory cytokine levels and intestinal flora in a self-healing dextran sodium sulfate-induced ulcerative colitis murine model
- Evaluation of Lacticaseibacillus casei 39 Paraprobiotic in Modulating Inflammatory and Oxidative Pathways in Acetic Acid Induced Ulcerative Colitis
- Fusobacterium nucleatum Extracellular Vesicles Promote Experimental Colitis by Modulating Autophagy via the miR-574-5p/CARD3 Axis
- Lacticaseibacillus casei IB1 Alleviates DSS-Induced Inflammatory Bowel Disease by Regulating the Microbiota and Restoring the Intestinal Epithelial Barrier
- Lactobacillus brevis Alleviates DSS-Induced Colitis by Reprograming Intestinal Microbiota and Influencing Serum Metabolome in Murine Model