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Salmonella
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Cell death in gut cells dependent on caspase-3 and caspase-7 activity affects Salmonella growth in mice. Staurosporine or doxorubicin causes cell death and increases Salmonella growth, even though doxorubicin usually kills bacteria. This shows that specific cell death processes in the gut are important for Salmonella to thrive.
In a conventional mouse model, Salmonella must directly compete with commensal microbiota for a nutritional niche
Sommer et al. 2017 Na…
References (Sources)
- Commensal Enterobacteriaceae protect against Salmonella colonization through oxygen competition
- Consistent activation of the beta-catenin pathway by Salmonella type-three secretion effector protein AvrA in chronically infected intestine
- Depletion of Butyrate-Producing Clostridia from the Gut Microbiota Drives an Aerobic Luminal Expansion of Salmonella
- Microbes exploit death-induced nutrient release by gut epithelial cells
- The resilience of the intestinal microbiota influences health and disease