Salmonella

Cell death in gut cells dependent on caspase-3 and caspase-7 activity affects Salmonella growth in mice. Staurosporine or doxorubicin causes cell death and increases Salmonella growth, even though doxorubicin usually kills bacteria. This shows that specific cell death processes in the gut are important for Salmonella to thrive.
Anderson et al. 2021 Nature 596: 262

In a conventional mouse model, Salmonella must directly compete with commensal microbiota for a nutritional niche
Sommer et al. 2017 Nat. Rev. Microbiol. 15: 630-638

Salmonella belongs to pathogenic bacteria. Their Lipopolysaccharides (LPS) containing membrane fragments induce as endotoxins enteritis going along with diarrhea (e.g., typhus). Using a mouse model, Salmonella colitis increases epithelial oxygenation, thereby driving an aerobic pathogen expansion in the gut lumen. These results elucidate a mechanism that increases oxygen avaiability in the lumen of large intestine during intestinal inflammation or after antibiotic treatment
Rivera-Chávez et al. 2016 Cell Host Microbe 19: 443

Salmonella use a molecular “needle,” referred to as a type-three secretion system, to inject bacterial effector proteins (e.g., AvrA) into host cells
Lu et al. 2012 Am. J. Physiol. Gastrointest. Liver Physiol. 303: G1113–G1125

see also:
Diarrhea / Diarrhoea & Salmonella enterica enterica Typhimurium
Infections & Salmonella / Salmonella typhimurium